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While we uncovered suppression of AGXT and induction of LDHA as the underlying mechanisms for oxalate overproduction in MASH, the possibility of altered ratios of cofactors for these and other enzymatic reactions (for example, GRHPR) that regulate glyoxylate/oxalate metabolism in MASH warrants further investigation

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Figure 2 In summary, Metabolomics studies have consistently demonstrated that the intestinal metabolite profiles of IBD patients differ significantly from those of healthy controls, including alterations in short-chain fatty acids (SCFAs), bile acids, and tryptophan-derived metabolites, which are repeatedly observed across independent cohorts (177, 178)